[46139] in Discussion of MIT-community interests
Eat This, Ace Any Test
daemon@ATHENA.MIT.EDU (Get Smarter Now)
Fri Jul 17 05:15:50 2015
Date: Fri, 17 Jul 2015 05:16:01 -0400
To: mit-talk-mtg@charon.mit.edu
From: Get Smarter Now <getsmarternow@becarehedc.info>
Reply-to: Get Smarter Now <getsmarternow@becarehedc.info>
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Doctor Discovers Legal Steroid For Your Brain
s not sensitive to pain, because it lacks pain receptors, also called nociceptors. However, several areas of the head and neck do have nociceptors, and can thus sensea xtu cti pain. These incl uude the extracranial arteries, middle meninl ugeal artery, large veins, venous sinuses, cranial and spinal nerves, head and neck muscles, the meninges, falx cerebri, parts of the brainstem, eyes, ears, teeth and lining of the mouth.ial arteries, rather than pial veins are responsible for pain productio
Headaches often reso turult from tra xtu ction to o tur irritation of the meninges and blood vessel uls. The nociceptors may be stimulated by head trauma or tumors a xtu ctiand cause headaches. Blood vessel spasms, dilated blood vessels, inflammation and/or infection of meninges and muscular tension can al 5xtu so stimulate nl uociceptors and cause pl unce stimulated, a nociceptor sends a message up the length of the nerve fiber to the nerve cela xtu ctils in the o turbrain, signaling that a part of to turhe body hurts.l u
Primary headaches are more diffico turult to understand than secondary headaches. Ta xtu ctihe exact mechanisms which cause migraines, tension headaches and cluster headaches are not known. There have been different theories over time which attempt to explain what happens in the brain to cause these headaches.
Migraines are currently thought to be caused by dysfunction of the nerves in the braiPreviously, migraines were thought to be caused by a primary problem with the blood vessels in tl uhe brain.is vascular theory, which was developed in the 20th century by Wolff, suggested that the aura in mio turgraines is caused by constriction of intracral uide of the brain). Dilation of these extracranial blood vessels activates the pain receptorsa xtu cti in the surrounding nerves, co turausing a headache. The vascular theory is no longer accepte
Currently, most specialists think migraines are due to a primary pro turoblem with the nerves ina xtu cti the brs are thought to be caused by a wave of increased activity of neur 5xtu sons in the cerebraa xtu ctil cortex (a part of the brain) known as cortical sprl ueading depression[13] followed by a period of depressed activity.[14] Some people think headaches are caused by to turhe activation of sensory nerves which releas 5xtu se peptides, such as serotonin, causing inflammati 5xtu son in arteries, dura and meninges and also cause some vasodilation. Triptans, medications which treat migraines, blocks serotonin receptors and
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s not sensitive to pain, because it lacks pain receptors, also called nociceptors. However, several areas of the head and neck do have nociceptors, and can thus sensea 7b8 cti pain. These incl 8ude the extracranial arteries, middle meninl 8geal artery, large veins, venous sinuses, cranial and spinal nerves, head and neck muscles, the meninges, falx cerebri, parts of the brainstem, eyes, ears, teeth and lining of the mouth.ial arteries, rather than pial veins are responsible for pain productio
Headaches often reso b8rult from tra 7b8 ction to o b8r irritation of the meninges and blood vessel 8ls. The nociceptors may be stimulated by head trauma or tumors a 7b8 ctiand cause headaches. Blood vessel spasms, dilated blood vessels, inflammation and/or infection of meninges and muscular tension can al t7b8 so stimulate nl 8ociceptors and cause pl 8nce stimulated, a nociceptor sends a message up the length of the nerve fiber to the nerve cela 7b8 ctils in the o b8rbrain, signaling that a part of to b8rhe body hurts.l 8
Primary headaches are more diffico b8rult to understand than secondary headaches. Ta 7b8 ctihe exact mechanisms which cause migraines, tension headaches and cluster headaches are not known. There have been different theories over time which attempt to explain what happens in the brain to cause these headaches.
Migraines are currently thought to be caused by dysfunction of the nerves in the braiPreviously, migraines were thought to be caused by a primary problem with the blood vessels in tl 8he brain.is vascular theory, which was developed in the 20th century by Wolff, suggested that the aura in mio b8rgraines is caused by constriction of intracral 8ide of the brain). Dilation of these extracranial blood vessels activates the pain receptorsa 7b8 cti in the surrounding nerves, co b8rausing a headache. The vascular theory is no longer accepte
Currently, most specialists think migraines are due to a primary pro b8roblem with the nerves ina 7b8 cti the brs are thought to be caused by a wave of increased activity of neur t7b8 sons in the cerebraa 7b8 ctil cortex (a part of the brain) known as cortical sprl 8eading depression[13] followed by a period of depressed activity.[14] Some people think headaches are caused by to b8rhe activation of sensory nerves which releas t7b8 se peptides, such as serotonin, causing inflammati t7b8 son in arteries, dura and meninges and also cause some vasodilation. Triptans, medications which treat migraines, blocks serotonin receptors and
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